Xanya Sofra-Weiss, Ph.D
Individual phenotypic differences result in a variation of T4 to Free T3 conversion. Free T3 stimulates lipolysis. This leads to polymorphic and individualized lipid deposition patterns. Hyperthyroidism is associated with weight loss via an increase in metabolic rate and lipolysis. Hypothyroidism, on the other hand, is associated with weight gain via a decrease in metabolic rate. A literature review by Guillermo et al (2003) has shown that the risk of thyroid dysfunction in Diabetic patients is two- to threefold higher than in the general population. A number of studies have shown that thyroid hormones represented by serum total T3 and T4 concentrations and serum Free T3 and T4 concentrations were significantly lower in obese non-insulin-dependent diabetics than control subjects. Low T3 is also a strong predictor of mortality in cardiac patients and may be directly implicated in the poor prognosis of cardiac patients. Although the benefits of intensified insulin treatment in insulin-dependent Diabetes Mellitus are well recognized, a meta-analysis of 14 randomized controlled trials revealed the risk of severe Hypoglycemia, Ketoacidosis and mortality from acute metabolic causes with intensified insulin treatment. These 14 trails contributed 16 comparisons with 1028 patients allocated to intensiļ¬ed and 1039 allocated to conventional treatment. A total of 846 patients suffered at least one episode of severe hypoglycaemia, 175 patients experienced ketoacidosis and 26 patients died. We are investigating an alternative treatment for Diabetes with no side effects. This involves the enhancement of endogenous production of Free T3 and IGF-1 via an electronically designed ionic signal. The mechanism of this therapeutic signal delivery was invented by Pollock, (1990-2008), in Innovations Science, a European Community-funded research center. Using the Pacemaker technology this ionic signal produces the physiological responses associated with strenuous exercise. Pollock’s ionic signal (image 1) initially targets the motor neurons resulting in rhythmical muscle contractions equivalent to performing high resistance physical activity. Once the process is initiated by Pollock’s bio-identical electronic signal, the motor neurons signal the brain via the spinal cord. This is a physiologically Beyond Bio-identical Hormones Ongoing Research on A New Potential Treatment for Adult reversed process, like traffic being driven the opposite way, where the strenuous exercise signal does not originate in the brain traveling down the spinal cord to the motor nerve. Instead, the process is initiated at the peripheral motor neuron, then the circuit is completed by outgoing CNS neuron emission. This CNS emission causes the ultimate production of Free T3 and GH/IGF-1, which in turn cause lipolysis and muscular hypertrophy. The enhanced production of Free T3 and GF/IGF-I will temporarily cause hyperglycemia. However, the hyperglycemia will resolve once the glucose has been utilized for metabolic purposes including increased cellular energy and muscular hypertrophy. The aim of this study is to test the hypothesis that the use of ionic currents may reduce or eliminate adult onset Diabetes.
Xanya Sofra Weiss
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